ncbi reference sequence database release 36 Search Results


rest  (Thermo Fisher)
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pro vstyr  (Thermo Fisher)
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36 musmusculus  (Biotechnology Information)
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zatebradine  (Tocris)
94
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Effect of CIL, ivabradine (IVA), or <t>zatebradine</t> (ZAT) on depolarization-elicited I K(DR) densities recorded from H9c2 cardiac cells. Cells were bathed in Ca 2+ -free Tyrode’s solution containing 1 μM tetrodotoxin, and we backfilled the recording pipette with a K + -containing solution. In ( A )¸ I K(DR) density elicited by 300-msec step depolarization from −50 to +50 mV was obtained in control and after addition of 1 μM CIL or 3 μM CIL, while in ( B ), that was taken in the absence and presence of 1 μM ZAT or 3 μM ZAT. The upper part in (A) or (B) is the voltage protocol used. ( C ) Summary bar graph of inhibitory effects of CIL (3 μM), IVA (3 μM), or ZAT (3 μM) on percentage decrease of I K(DR) in H9c2 cells (mean ± SEM; n = 8). The current density was measured at the end of the 300-msec depolarizing pulse from −50 to +50 mV. * Significantly different from CIL (3 μM) group ( p < 0.05).
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ncbi protein database  (Biotechnology Information)
86
Biotechnology Information ncbi protein database
Effect of CIL, ivabradine (IVA), or <t>zatebradine</t> (ZAT) on depolarization-elicited I K(DR) densities recorded from H9c2 cardiac cells. Cells were bathed in Ca 2+ -free Tyrode’s solution containing 1 μM tetrodotoxin, and we backfilled the recording pipette with a K + -containing solution. In ( A )¸ I K(DR) density elicited by 300-msec step depolarization from −50 to +50 mV was obtained in control and after addition of 1 μM CIL or 3 μM CIL, while in ( B ), that was taken in the absence and presence of 1 μM ZAT or 3 μM ZAT. The upper part in (A) or (B) is the voltage protocol used. ( C ) Summary bar graph of inhibitory effects of CIL (3 μM), IVA (3 μM), or ZAT (3 μM) on percentage decrease of I K(DR) in H9c2 cells (mean ± SEM; n = 8). The current density was measured at the end of the 300-msec depolarizing pulse from −50 to +50 mV. * Significantly different from CIL (3 μM) group ( p < 0.05).
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dimethyl phthalate  (Thermo Fisher)
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Effect of CIL, ivabradine (IVA), or <t>zatebradine</t> (ZAT) on depolarization-elicited I K(DR) densities recorded from H9c2 cardiac cells. Cells were bathed in Ca 2+ -free Tyrode’s solution containing 1 μM tetrodotoxin, and we backfilled the recording pipette with a K + -containing solution. In ( A )¸ I K(DR) density elicited by 300-msec step depolarization from −50 to +50 mV was obtained in control and after addition of 1 μM CIL or 3 μM CIL, while in ( B ), that was taken in the absence and presence of 1 μM ZAT or 3 μM ZAT. The upper part in (A) or (B) is the voltage protocol used. ( C ) Summary bar graph of inhibitory effects of CIL (3 μM), IVA (3 μM), or ZAT (3 μM) on percentage decrease of I K(DR) in H9c2 cells (mean ± SEM; n = 8). The current density was measured at the end of the 300-msec depolarizing pulse from −50 to +50 mV. * Significantly different from CIL (3 μM) group ( p < 0.05).
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interpro dataset v.53  (InterPro Inc)
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Effect of CIL, ivabradine (IVA), or <t>zatebradine</t> (ZAT) on depolarization-elicited I K(DR) densities recorded from H9c2 cardiac cells. Cells were bathed in Ca 2+ -free Tyrode’s solution containing 1 μM tetrodotoxin, and we backfilled the recording pipette with a K + -containing solution. In ( A )¸ I K(DR) density elicited by 300-msec step depolarization from −50 to +50 mV was obtained in control and after addition of 1 μM CIL or 3 μM CIL, while in ( B ), that was taken in the absence and presence of 1 μM ZAT or 3 μM ZAT. The upper part in (A) or (B) is the voltage protocol used. ( C ) Summary bar graph of inhibitory effects of CIL (3 μM), IVA (3 μM), or ZAT (3 μM) on percentage decrease of I K(DR) in H9c2 cells (mean ± SEM; n = 8). The current density was measured at the end of the 300-msec depolarizing pulse from −50 to +50 mV. * Significantly different from CIL (3 μM) group ( p < 0.05).
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ncb-ph  (BARCO Inc)
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Effect of CIL, ivabradine (IVA), or <t>zatebradine</t> (ZAT) on depolarization-elicited I K(DR) densities recorded from H9c2 cardiac cells. Cells were bathed in Ca 2+ -free Tyrode’s solution containing 1 μM tetrodotoxin, and we backfilled the recording pipette with a K + -containing solution. In ( A )¸ I K(DR) density elicited by 300-msec step depolarization from −50 to +50 mV was obtained in control and after addition of 1 μM CIL or 3 μM CIL, while in ( B ), that was taken in the absence and presence of 1 μM ZAT or 3 μM ZAT. The upper part in (A) or (B) is the voltage protocol used. ( C ) Summary bar graph of inhibitory effects of CIL (3 μM), IVA (3 μM), or ZAT (3 μM) on percentage decrease of I K(DR) in H9c2 cells (mean ± SEM; n = 8). The current density was measured at the end of the 300-msec depolarizing pulse from −50 to +50 mV. * Significantly different from CIL (3 μM) group ( p < 0.05).
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candida utilis atcc 9950  (ATCC)
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ATCC candida utilis atcc 9950
Effect of CIL, ivabradine (IVA), or <t>zatebradine</t> (ZAT) on depolarization-elicited I K(DR) densities recorded from H9c2 cardiac cells. Cells were bathed in Ca 2+ -free Tyrode’s solution containing 1 μM tetrodotoxin, and we backfilled the recording pipette with a K + -containing solution. In ( A )¸ I K(DR) density elicited by 300-msec step depolarization from −50 to +50 mV was obtained in control and after addition of 1 μM CIL or 3 μM CIL, while in ( B ), that was taken in the absence and presence of 1 μM ZAT or 3 μM ZAT. The upper part in (A) or (B) is the voltage protocol used. ( C ) Summary bar graph of inhibitory effects of CIL (3 μM), IVA (3 μM), or ZAT (3 μM) on percentage decrease of I K(DR) in H9c2 cells (mean ± SEM; n = 8). The current density was measured at the end of the 300-msec depolarizing pulse from −50 to +50 mV. * Significantly different from CIL (3 μM) group ( p < 0.05).
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bluegnome cytochip 2.01  (BlueGnome Limited)
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Effect of CIL, ivabradine (IVA), or <t>zatebradine</t> (ZAT) on depolarization-elicited I K(DR) densities recorded from H9c2 cardiac cells. Cells were bathed in Ca 2+ -free Tyrode’s solution containing 1 μM tetrodotoxin, and we backfilled the recording pipette with a K + -containing solution. In ( A )¸ I K(DR) density elicited by 300-msec step depolarization from −50 to +50 mV was obtained in control and after addition of 1 μM CIL or 3 μM CIL, while in ( B ), that was taken in the absence and presence of 1 μM ZAT or 3 μM ZAT. The upper part in (A) or (B) is the voltage protocol used. ( C ) Summary bar graph of inhibitory effects of CIL (3 μM), IVA (3 μM), or ZAT (3 μM) on percentage decrease of I K(DR) in H9c2 cells (mean ± SEM; n = 8). The current density was measured at the end of the 300-msec depolarizing pulse from −50 to +50 mV. * Significantly different from CIL (3 μM) group ( p < 0.05).
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gene expression omnibus  (Biotechnology Information)
86
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Effect of CIL, ivabradine (IVA), or <t>zatebradine</t> (ZAT) on depolarization-elicited I K(DR) densities recorded from H9c2 cardiac cells. Cells were bathed in Ca 2+ -free Tyrode’s solution containing 1 μM tetrodotoxin, and we backfilled the recording pipette with a K + -containing solution. In ( A )¸ I K(DR) density elicited by 300-msec step depolarization from −50 to +50 mV was obtained in control and after addition of 1 μM CIL or 3 μM CIL, while in ( B ), that was taken in the absence and presence of 1 μM ZAT or 3 μM ZAT. The upper part in (A) or (B) is the voltage protocol used. ( C ) Summary bar graph of inhibitory effects of CIL (3 μM), IVA (3 μM), or ZAT (3 μM) on percentage decrease of I K(DR) in H9c2 cells (mean ± SEM; n = 8). The current density was measured at the end of the 300-msec depolarizing pulse from −50 to +50 mV. * Significantly different from CIL (3 μM) group ( p < 0.05).
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promoterinspector program eldorado 08-2006  (Genomatix gmbh)
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Effect of CIL, ivabradine (IVA), or <t>zatebradine</t> (ZAT) on depolarization-elicited I K(DR) densities recorded from H9c2 cardiac cells. Cells were bathed in Ca 2+ -free Tyrode’s solution containing 1 μM tetrodotoxin, and we backfilled the recording pipette with a K + -containing solution. In ( A )¸ I K(DR) density elicited by 300-msec step depolarization from −50 to +50 mV was obtained in control and after addition of 1 μM CIL or 3 μM CIL, while in ( B ), that was taken in the absence and presence of 1 μM ZAT or 3 μM ZAT. The upper part in (A) or (B) is the voltage protocol used. ( C ) Summary bar graph of inhibitory effects of CIL (3 μM), IVA (3 μM), or ZAT (3 μM) on percentage decrease of I K(DR) in H9c2 cells (mean ± SEM; n = 8). The current density was measured at the end of the 300-msec depolarizing pulse from −50 to +50 mV. * Significantly different from CIL (3 μM) group ( p < 0.05).
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Image Search Results


Effect of CIL, ivabradine (IVA), or zatebradine (ZAT) on depolarization-elicited I K(DR) densities recorded from H9c2 cardiac cells. Cells were bathed in Ca 2+ -free Tyrode’s solution containing 1 μM tetrodotoxin, and we backfilled the recording pipette with a K + -containing solution. In ( A )¸ I K(DR) density elicited by 300-msec step depolarization from −50 to +50 mV was obtained in control and after addition of 1 μM CIL or 3 μM CIL, while in ( B ), that was taken in the absence and presence of 1 μM ZAT or 3 μM ZAT. The upper part in (A) or (B) is the voltage protocol used. ( C ) Summary bar graph of inhibitory effects of CIL (3 μM), IVA (3 μM), or ZAT (3 μM) on percentage decrease of I K(DR) in H9c2 cells (mean ± SEM; n = 8). The current density was measured at the end of the 300-msec depolarizing pulse from −50 to +50 mV. * Significantly different from CIL (3 μM) group ( p < 0.05).

Journal: International Journal of Molecular Sciences

Article Title: Inhibitory Effective Perturbations of Cilobradine (DK-AH269), A Blocker of HCN Channels, on the Amplitude and Gating of Both Hyperpolarization-Activated Cation and Delayed-Rectifier Potassium Currents

doi: 10.3390/ijms21072416

Figure Lengend Snippet: Effect of CIL, ivabradine (IVA), or zatebradine (ZAT) on depolarization-elicited I K(DR) densities recorded from H9c2 cardiac cells. Cells were bathed in Ca 2+ -free Tyrode’s solution containing 1 μM tetrodotoxin, and we backfilled the recording pipette with a K + -containing solution. In ( A )¸ I K(DR) density elicited by 300-msec step depolarization from −50 to +50 mV was obtained in control and after addition of 1 μM CIL or 3 μM CIL, while in ( B ), that was taken in the absence and presence of 1 μM ZAT or 3 μM ZAT. The upper part in (A) or (B) is the voltage protocol used. ( C ) Summary bar graph of inhibitory effects of CIL (3 μM), IVA (3 μM), or ZAT (3 μM) on percentage decrease of I K(DR) in H9c2 cells (mean ± SEM; n = 8). The current density was measured at the end of the 300-msec depolarizing pulse from −50 to +50 mV. * Significantly different from CIL (3 μM) group ( p < 0.05).

Article Snippet: Cilobradine (CIL, DK-AH269, 3-[[(3S)-1-[2-(3,4-dimethoxyphenyl)ethyl]-3-piperidinyl]methyl]-1,3,4,5-tetrahydro-7,8-dimethoxy-2H-3-benzazepin-2-one, monohydrochloride, C 28 H 38 N 2 O 5 ·HCl, https://pubchem.ncbi.nlm.nih.gov/compound/16078969 ) was acquired from Cayman Chemical (Excel Biomedical, Taipei, Taiwan), and tefluthrin (Tef), tetraethylammonium chloride (TEA), tetrodotoxin, and ivabradine (IVA, https://pubchem.ncbi.nlm.nih.gov/compound/Ivabradine ) were from Sigma-Aldrich (Merck Ltd., Taipei, Taiwan), while A-803467 (5-(4-chlorophenyl)-N-(3,5-dimethoxyphenyl)furan-2-carboxamide) and zatebradine (ZAT, UL-FS49, C 26 H 36 N 2 O 5 , https://pubchem.ncbi.nlm.nih.gov/compound/Zatebradine ) were from Tocris (Union Biomed Inc., Taipei, Taiwan).

Techniques: Transferring, Control